無水斷(Dry fasting)我是參考Gavin McGowen。無水斷效果：1天無水斷=3天有水斷=6天果汁斷。我喜歡效果快一點，而且對我而言，無水斷比有水斷容易(完全不會引起食慾) 且省好多時間，不用想吃喝的事，無水斷12~24小時就可以達到ketosis，比起有水斷72小時甚至1星期快很多。
請問無水斷74小時後 你會如何復時?是先水斷一段間 然後再吃嗎? 有復胖嗎?
先復水半天到一天，然後喝骨頭湯， 之後就是低醣飲食(30g), 只要保持尿生酮180(++++),體重就會一直下降，而且掉很快， 尤其身體變成燒脂機器後， 就不會有想吃醣的"癮"。達成體重目標後， 就可開始加入水果， 澱粉蔬菜和偶爾的穀類, (100g) /天之內， 都可保持體重。這已變成生活的習慣，別把它當成一時的減肥方式。不管任何減重方式， 如果又回到高澱粉飲食， 那註定會失敗。
DRY FASTING: THE ULTIMATE PATH TO LONGEVITY
A TRUE LIFE OR DEATH SCENARIO
On extended dry fasts the first few days are the roughest as the body is still optimistically holding out for food or water but once it figures out that water and food are not coming and that death is imminent if it doesn't adapt and fast to given circumstance it switches gears into survival mode— this highly adapted state IS the magical key of dry fasting. Survival of the fittest. All weakness is eradicated. The option of supporting the diseased and parasitic is no longer viable. All non essentials are eliminated and recycled to sustain the essentials. This IS cellular renewal at the deepest level— unattainable by any other means.
A 5 DAY
DRY FAST SCIENTIFIC STUDY:
While indeed longer duration dry fasts, 3 to 7 days and beyond, are best utilized to bring an ill or obese person back into balance—once homeostasis is restored daily dry fasting is the most effective disease preventive measure one can employ in maintaining optimal health and delaying aging.
This is the how and why behind dry fasting.
DAILY DRY FASTING in conjunction with anti-aging herbs and diet— the SCIENCE behind it.
daily dry fasting which is your wisest protocol for maintaining a
precision tuned body built for the long haul a 1-6 hour eating
window is recommended with your highest level of activity including
workout around this time as well.
RESTRICTION OF FEEDING TIME —WHY?
**It's not what you eat so much as WHEN you eat. **
"Feeding schedule has been shown to have a signifiant impact on
health and survival. In studies, time-restricted feeding had
profound effects on neural, peripheral, and cardiovascular
physiology and improved sleep, body weight maintenance, and delayed
signs of cardiac aging, under UNCHANGED caloric intake and
are 3 factors we are focusing on that promotes longevity and absence
All 3 are activated by KETOSIS* which is a rapid result of DRY FASTING. (No water or food.)
BODIES STIMULATE CHAPERONE-MEDIATED AUTOPHAGY
these studies we have demonstrated that ketone bodies, more
specifically BOH, stimulate CMA by causing the oxidation of
substrates. In addition, during prolonged starvation CMA is
activated because of increased lamp2a in the lysosomal membrane and
increased lyhsc70 in the lysosomal lumen (9, 31). Our data indicate
that ketone bodies can also stimulate CMA by affecting substrate
proteins during prolonged starvation in vivo. This finding gives us
further insight into the physiological importance of CMA stimulation
during times of nutrient deprivation."
Chaperone-mediated autophagy: roles in disease and aging http://www.nature.com/cr/journal/v24/n1/pdf/cr2013153a.pdf
EXCELLENT VIDEO visually explaining the 3 types of AUTOPHAGY:
Question: If dehydration RAISES blood sugar and fasting LOWERS blood sugar —what happens when you combine them together as in dry fasting?
ULTRA RAPID ONSET OF KETOSIS! :)
"Fasting resulted in reduced plasma glucose concentrations compared with the control study, while dehydration resulted in increased plasma glucose concentrations compared with the control study (P < .001). Glucose production and disposal were decreased during the fasting study and increased during the dehydration study compared with the control study.
***Glucagon concentrations and rates of development of ketosis and metabolic acidosis were increased during both fasting and dehydration compared with control.***
data suggest that fasting and dehydration have differential effects
on glycemia during insulin deficiency, with dehydration favoring the
development of hyperglycemia and fasting resulting in reduced
KETOSIS important and what is it's link to autophagy, mTOR
inhibition, AMPK activation?
restriction and its effects on aging:
All about mTOR: http://selfhacked.com/…/11/03/mtor-natural-mtor-inhibitors/…
AMPK has been referred to as a “metabolic master switch.” AMPK controls a gamut of metabolic pathways that enable us to extract energy from food, store and distribute that energy safely through the body, and ultimately use that energy for everything from moving and mating to talking and thinking, and even to understanding these very words as you read them.
The core role of AMPK is to sense each cell’s energy status at every moment, and to trigger responses that maintain the cell’s energy at precisely the optimum level. Too little available energy starves the cell, while too much energy can exhaust and disrupt cellular components.In either case (too little or too much energy), the cell (and the tissues, organs, and systems in which it is a part) functions inefficiently. That energy inefficiency ultimately leads to the dysfunctions we identify as the diseases (or symptoms) of aging.
Here’s how AMPK works: Every cell in your body depends absolutely on a steady supply of energy in the form of chemical bonds. When you eat and absorb nutrients, energy from chemical bonds in food is released and passed down a complex series of enzymes until it is stored again in a molecule called adenosine triphosphate, or ATP. The more ATP that is present in the cell, the higher the cell’s available energy supply. When ATP is broken down to release energy for cellular work, a major end product is adenosine monophosphate, or AMP.
In preclinical research, enhanced AMPK activity has been associated with a 20-30% increase in life span, but that’s just the beginning of the health benefits conferred by this critical cellular enzyme.
Increased AMPK activation has been shown to help reduce fat storage (especially dangerous belly fat), increase insulin sensitivity (to lower blood glucose), reducecholesterol/triglyceride production, and suppress chronic inflammation. All of these factors underlie the lethal diseases of aging."
Autophagy is the best Way to Get Rid of (Cellular) Junk. READ: http://www.anti-agingfirewalls.com/…/autophagy-the-houseke…/
AUTOPHAGY regulates AGING:
AMPK and autophagy also are activated by intense exercise. More here: https://scholar.google.com/scholar…
Exercise ENHANCES rather than inhibits mTOR and increases protein synthesis (muscle growth) so you want to time it near feeding window. More here: https://scholar.google.com/scholar…
"There is overwhelming evidence that cellular mechanisms and signaling pathways regulating ageing are controlled by mTOR. Here we have highlighted important studies that support a role for both mTOR dependent protein synthesis and autophagy in ageing."
DRY FAST TO INHIBIT MTOR***
The LESS you keep the MTOR pathway open or active the GREATER your health will be and the LONGER you will live.
Growth or longevity. One or the other.
Here's your percentages of mTOR activation based on feeding and intense exercise window times:
window is 25%
The smaller the window the better.
Don't dilly dally when it's open.
When the feeding window and MTOR pathway is OPEN it's GO TIME. You go into the gym and WRECK SHOP, refuel and rehydrate then close the window and shut down mTOR again.
AMPK ACTIVATION AND INTENSE INTERVAL EXERCISE
"Brief intense interval exercise activates AMPK and p38 MAPK signaling and increases the expression of PGC-1 in human skeletal muscle
summary, the present study showed that four 30-s bouts of all out
cycling increased phosphorylation of AMPK 1, AMPK 2, and p38 MAPK
immediately following exercise and the mRNA expression of PGC-1
after 3 h of recovery. Specific signaling through AMPK and p38 MAPK
to PGC-1 may therefore explain in part the metabolic remodeling in-
duced by intense interval exercise training, including mitochondrial
biogenesis and an increased capacity for glucose and fatty acid
ACTIVATES mTOR and thus increases protein synthesis so it can be
used to accelerate muscle growth AT BEGINNING of exercise window
"That which doesn't kill you makes you stronger." — Nietzsche
"All things are poison and nothing is without poison; only the dose makes a thing not a poison." —Paracelsus
Even water and oxygen in excess can be deadly; as can their absence.
What we are doing is deliberately stressing the body— to make it more resilient.
"Hormesis is a biological phenomenon whereby a beneficial effect (improved health, stress tolerance, growth or longevity) results from exposure to low doses of an agent that is otherwise toxic or lethal when given at higher doses."
Too much dry fasting can kill just as over eating or over drinking can kill.
We are in a sense flexing our cells "muscles"; making them extremely adaptable and tough.
"No water? No food? No problem. Wake me up when we got a real crisis on our hands."
All about hormesis here: https://scholar.google.com/scholar…
***INSULIN BLOCKS SIRT1*** (not good)
SIRT1 stands for sirtuin (silent mating type information regulation 2 homolog) 1 (S. cerevisiae), referring to the fact that its sirtuin homolog (biological equivalent across species) in yeast (S. cerevisiae) is Sir2. SIRT1 is an enzyme that deacetylates proteins that contribute to cellular regulation (reaction to stressors, longevity).
cause of aging is thought to result from the cumulative effects of
cell loss over time. In yeast, caloric restriction (CR) delays aging
by activating the Sir2 deacetylase. Here we show that expression of
mammalian Sir2 (SIRT1) is induced in CR rats as well as in human
cells that are treated with serum from these animals. Insulin and
insulin-like growth factor 1 (IGF-1) attenuated this response. SIRT1
deacetylates the DNA repair factor Ku70, causing it to sequester the
proapoptotic factor Bax away from mitochondria, thereby inhibiting
stress-induced apoptotic cell death. Thus, CR could extend life-span
by inducing SIRT1 expression and promoting the long-term survival of
AND STEM CELL REGENERATION
system defects are at the center of aging and a range of diseases.
Here, we show that prolonged fasting reduces circulating IGF-1
levels and PKA activity in various cell populations, leading to
signal transduction changes in long-term hematopoietic stem cells
(LT-HSCs) and niche cells that promote stress resistance,
self-renewal, and lineage-balanced regeneration. Multiple cycles of
fasting abated the immunosuppression and mortality caused by
chemotherapy and reversed age-dependent myeloid-bias in mice, in
agreement with preliminary data on the protection of lymphocytes
from chemotoxicity in fasting patients. The proregenerative effects
of fasting on stem cells were recapitulated by deficiencies in
either IGF-1 or PKA and blunted by exogenous IGF-1. These findings
link the reduced levels of IGF-1 caused by fasting to PKA signaling
and establish their crucial role in regulating hematopoietic stem
cell protection, self-renewal, and regeneration.
METABOLIC TRIGGERS OF AUTOPHAGY
RAMADAN IS DAILY INTERMITTENT DRY FASTING —why not apply this wise practice every day and not just Ramadan?
"Fasting in the month of Ramadan is ordained on the Muslim believers. Ramadan fasting has not only been spiritually beneficial but it has physical, psychological, social and health benefits.
Ramadan is a month of self regulation, process of self purification, truthfulness and self trainings with the hope that this training will last beyond the end of Ramadan. One advantage of fasting is that the poor are given attention and benefits from charity and the faithful practice of the concept of neighbourhood and hospitality.
The physiological effect of fasting includes lowering of blood sugar, lowering of cholesterol and lowering of the systolic blood pressure. In fact, Ramadan fasting would be an ideal recommendation for treatment of mild to moderate, stable, non-insulin diabetes, obesity and essential hypertension. Fasting is powerful therapeutic processes that can help people recover from mild to severe health conditions. Our body has a self healing power, in order to activate this power, the stomach must be kept empty, if Ramadan fasting is done properly it can help to recover from most diseases."
P53: THE ANTI-TUMOR GENE AND FASTING
findings that CR or a 1 day/week fast suppressed carcinogenesis—even
when started late in life in mice predestined to develop tumors due
to decreased p53 gene dosage—support
efforts to identify suitable interventions influencing energy
balance in humans as a tool for cancer prevention."
the anti-aging hunger hormone is released about 12 hours after your
last meal. This is why you want lots of Ghrelin:
what about lack of water and kidneys?
PROTECT KIDNEYS by ALKALINIZING periodically before and after dry fasts with a simple teaspoon of BAKING SODA in water.
Hydration and Alkalinization is Noninferior to Intravenous Therapy
for Prevention of Contrast-Induced Nephropathy in Patients with
Chronic Kidney Disease"
HEADACHES are common on the first day for new dry fasters and those not experienced with KETOSIS. It usually takes place around the 18 hour mark and goes away after a few hours. This is the brain switching off glucose and now using ketones for fuel. The headache could also be symptomatic of caffeine withdrawal. Totally normal; push through, it passes.
INCREASE IN LIFESPAN
"Remarkably, the greatest increase in lifespan was observed
...maintained in the complete absence of food, with a 50% increase
in median and maximum lifespan relative to control-fed animals."
INCREASES LIFESPAN UP TO 60%
"Dietary Restriction (DR) increases the rodent lifespan by up to 60% in part by delaying the occurrence of many chronic diseases, and in part by slowing the rate of biological or physiological aging (1).
its many protective effects of DR described in mice, the most
notable include those on increasing insulin sensitivity and on the
attenuation of B amyloid deposition in a model for Alzheimer’s
Disease (54). Severe dietary restriction or STARVATION (dry fasting)
may also be applicable to disease treatment. For example, fasting
protects mice against high dose chemotherapy in part by reducing
serum IGF-I signalling but does not protect cancer cells, since
oncogene mutations prevent the activation of the stress resistance
pathways in response to the reduction in glucose, IGF-I and other
growth factors caused by nutrient deprivation (55)."
higher the metabolism speed, the shorter the lifespan, and vice
Over 24 weeks, a low-carbohydrate diet program led to greater weight loss, reduction in serum triglyceride level, and increase in HDL cholesterol level compared with a low-fat diet.
STARVATION TREATMENT OF DIABETES
(the starvation hormone) FASTING & LONGEVITY
FGF21 is specifically induced by HMGCS2 activity. The oxidized form of ketone bodies (acetoacetate) in a cultured medium also induced FGF21, possibly via a SIRT1-dependent mechanism. HMGCS2 activity has also been shown to be increased by deacetylation of lysines 310, 447, and 473 via SIRT3 in the mitochondria.
FGF21 is expressed in numerous tissues, including liver, brown
adipose tissue, white adipose tissue and pancreas, circulating
levels of FGF21 are derived specifically from the liver in mice.
In liver FGF21 expression is regulated by PPARα
and levels rise substantially with both FASTING and consumption of
ACID and the importance of minerals
CONCEPTUAL REVOLUTION OF BIBLICAL PROPORTIONS
constipation or bowel issues:
ACTIVATORS & MTOR INHIBITORS
in blend that inhibit mTOR:
Purchase INTERSTELLAR BLEND here: www.interstellarblend.com
REMEMBER: The less you drink the faster the fast works so if you do the morning drink keep it to 4-6 oz. Don't ingest anything else until feeding/exercise window.
WHY USE BAKING SODA FOR PREVENTING KIDNEY STONES OR KIDNEY PROBLEMS?
Because it works and fast!
I recommend raising urine ph to an 8 NOT 7 as in study before long dry fasts and once again after.
PH STRIPS TO TEST URINE: http://www.amazon.com/…/…/B00LY1KIWY/ref=zg_bs_3013605011_10
Conclusions: Bicarbonate therapy remains an attractive option for the treatment of radiolucent kidney stones. The presence of hyperuricaemia or hyperuricosuria appears to influence the success rate. Further prospective randomised studies are needed to identify the most tolerable and effective treatment regime as well as the optimal duration of treatment. Dual-energy CT may hold the key to identifying patients most likely to benefit from treatment.
ACTIVATORS/ MTOR INHIBITORS
THIRST AND HEAT
5. STUDY THIS POST AND THE LINKS. The more informed you are the less fear you will have and the greater resolve to not quit.
****IF YOU GOT FAT YOU GOT WATER.****
So have ZERO FEAR of going 3 days your first time. Alkalinize and launch!
skin tenting or skin turgor test???
Be not overly concerned by blood pressure irregularities; the body as it acclimates itself to survival conditions corrects itself the longer you go. It may fluctuate up and down the first 3 days but by day 4 and onward it stabilizes.
Blood pressure and resting pulse without fail on all extended dry fasts (beyond 3 days) goes down and this is very positive.
Have no fear or worries; perfectly natural process.
BUT WHAT ABOUT GLUCOSE REQUIREMENTS ON A DRY FAST AND THE LOSS OF MUSCLE ???
my muscles be broken down to create critical glucose required by
certain organs and red blood cells (that don't have mitochondria and
can't run on ketones) through the process of gluconeogenesis? Good
question. Let me ask you this: "If I designed the body and made it
to where under starvation conditions critical muscle was broken down
BEFORE fat would you think I was a very intelligent designer???" No
I didn't think so; that would be utterly ridiculous and foolish.
Glucose CAN and IS created from fat and here's how it works:
The human body with four billion years of evolution is far beyond our ability to abstraction, but in origin the body is relatively simple: everything comes, everything is soaked, and everything is governed by photosynthesis, both in plants and in us. http://www.ncbi.nlm.nih.gov/…/P…/pdf/1878-5085-5-S1-A146.pdf
THE HUMAN CHLOROPHYLL
GLUCONEOGENESIS FROM FAT
"That sugar can be converted into fatty acids in humans is a well-known fact. The question whether the reverse direction, i.e., gluconeogenesis from fatty acids, is also feasible has been a topic of intense debate since the end of the 19th century. With the discovery of the glyoxylate shunt that allows this conversion in some bacteria, plants, fungi and nematodes it has been considered infeasible in humans since the corresponding enzymes could not be detected. However, by this finding only a single route for gluconeogenesis from fatty acids has been ruled out. To address the question whether there might exist alternative routes in humans we searched for gluconeogenic routes from fatty acids in a metabolic network comprising all reactions known to take place in humans.
****Thus, we were able to identify several pathways showing that this conversion is indeed feasible.****
Analyzing evidence concerning the detected pathways lends support to
their importance during times of starvation, fasting, carbohydrate
reduced and ketogenic diets and other situations in which the
nutrition is low on carbohydrates. Moreover, the energetic
investment required for this pathway can help to explain the
particular efficiency of carbohydrate reduced and ketogenic diets
such as the Atkins diet."
Worry is misuse of your imagination; a form of self sabotage. Willfully replace all worry with joyous optimism and courage. Say to worry, "And if that perchance happens I will face it too gracefully and skillfully as I have many obstacles and challenges in my life thus far; I have no fear or worry. I am a fighter until the end. Now skedaddle worry! BE GONE FROM MY MIND. "
IDEAL FUEL FOR BODY
The energy producing factories of our cells – the mitochondria – work much better on a ketogenic diet as they are able to increase energy levels in a stable, long-burning, efficient, and steady way. Not only that, a ketogenic diet induces epigenetic changes which increases the energetic output of our mitochondria, reduces the production of damaging free radicals, and favours the production of GABA – a major inhibitory brain chemical. GABA has an essential relaxing influence and its favored production by ketosis also reduces the toxic effects of excitatory pathways in our brains. Furthermore, recent data suggests that ketosis alleviates pain in addition to having an overall anti-inflammatory effect. 
The ketogenic diet acts on multiple levels at once, something that no drug has been able to mimic. This is because mitochondria are specifically designed to use fat for energy. When our mitochondria use fat as an energetic source, its toxic load is decreased, the expression of energy producing genes are increased, its energetic output is increased, and the load of inflammatory energetic-end-products is decreased.
The key of these miraculous healing effects relies on the fact that fat metabolism and its generation of ketone bodies (beta-hydroxybutyrate and acetoacetate) by the liver can only occur within the mitochondrion, leaving chemicals within the cell but outside the mitochondria readily available to stimulate powerful anti-inflammatory antioxidants. The status of our mitochondria is the ultimate key for optimal health and while it is true that some of us might need extra support in the form of nutritional supplementation to heal these much needed energy factories, the diet still remains the ultimate key for a proper balance." More here: http://www.drmyhill.co.uk/…/Ketogenic_diet_-_a_connection_b…
RELEASES HUMAN GROWTH HORMONE
TRUE KETOSIS VS FAKE KETOSIS
True ketosis comes from starvation (fasting) or severe carbohydrate restriction. There are no high glucose levels under these conditions. The ketones come from the actual break down of body fat.
False ketosis is the result of coconut oil or mct oil. These provide ketones while being able to eat massive amounts of carbohydrates. The ketones are NOT coming from body fat but the coconut or mct oil. Yes ketostix will say you are in ketosis but you are really in fake ketosis.
You want TRUE not fake ketosis. Avoid coconut or mct oil until you are in TRUE ketosis and maintain less than 30g carbs a day.
IF you are at optimal weight and not trying to lose fat then yes coconut or mct oil and low glycemic load carbohydrates is not a problem.
*For someone on a KETOGENIC diet for epileptic purposes this gives them the advantage of more carbs. For someone trying to lose fat this is a disadvantage because your body will use the coconut oil ketones over body fat ketones for energy.
ALCOHOL STOPS FAT LOSS
The quickest way to halt weight loss and put on fat is alcohol. Why? Because alcohol is the body's first choice to burn for fuel. Guess what happens to any food you mix with it? That's right it goes straight to your waistline until the alcohol is burned off.
AVOID alcohol until you reach ideal weight. The fastest way to get over alcohol addiction is a 72 hour dry fast— your brain is purged clean with ZERO CRAVINGS.
HERES TONS OF ARTICLES ON IT: https://www.google.com/search…
PURE, WHITE AND DEADLY
The type of food recommended should be low glycemic load. Chart here: http://www.lowglycemicload.com/glycemic_table.html
You want minimal insulin spikes. Insulin and insulin resistance rapidly ages you. More here: https://scholar.google.com/scholar…
Vegan or non vegan isn't as important as balanced and nutrient dense. If you are vegan be vegan if you feel best with plants and meat do that. DO EAT PLANTS regardless.
Meat should only be eaten alone or with vegetables never starch or fruit. Salad, meat fish or chicken is a perfect pairing.
If vegan be sure to choose low glycemic load vegetables, fruits, nuts seeds and grains. http://www.lowglycemicload.com/glycemic_table.html (stay as close to 15 as possible)
KETO IDEAS: http://ketomotive.com/vegan-ketogenic-diet/
GLYCEMIC LOAD FRUITS:
about fruit juice fasting??
#2 Autophagy is induced by ketosis which is induced by fasting or extreme protein/carbohydrate restriction.
#3 A fast is NOT drinking a bunch of sugary bottled fruit juice which does NOT induce either ketosis or autophagy.
person that wants to sell you a "detox kit" ask them point blank,
"Does this inhibit mTOR, activate AMPK, promote autophagy or induce
Fructose which converts in liver to Glucose halts not only Ketosis but mTOR inhibition, AMPK activation AND autophagy (the whole point of fasting!!!) especially if you ingest more than 30g a day— thats about one 8oz glass of fruit juice. So instead of "juice feasting" all day long accomplishing nothing but wasting a bunch of money and overdosing on fructose an intelligent strategy would be to eat your fruit whole WITH IT'S FIBER (less blood sugar spikes) at the BEGINNING of your feeding window/pre workout.
Fruit without fiber is straight fructose.
Here's what's wrong with too much fructose:
Fructose Utilization and Associated Metabolic Dysfunction
"Consumption of fructose has been shown to be highly correlated with the development of diabetes, obesity and the metabolic syndrome. Consumption of soft drinks (high in HFCS) is associated with an increased risk for obesity in adolescents and for type 2 diabetes in young and middle-aged women. Excess fruit juice (also rich in fructose) is associated with the development of obesity in children.
Consumption of fructose by laboratory animals results in their
developing several features of metabolic syndrome, including
obesity, visceral fat accumulation, fatty liver, and elevated
insulin and leptin levels. It is likely that the increase in leptin
following fructose consumption represents leptin resistance, which
could account for the increased food intake observed in fructose-fed
high nitric oxide producing plants like beets and arugula. More on
nitric oxide and longevity here:
and its relationship with metabolism, calorie restriction, mTOR
inhibition, AMPK activation, autophagy and glucose:
ADDING SUGAR TO ANYTHING.
You DO NOT want your blood sugar spiking and releasing insulin. You want the highest insulin sensitivity possible. These Glucose Disposal Agents in conjunction with dry fasting can assist in regulating blood sugar.
BITTER MELON & ANTIDIABETIC AMPK ACTIVATION
Extracts of Fenugreek
17. Gynostemma Pentaphyllum (in Interstellar Blend)
"Gynostemma pentaphyllum is a plant distantly related to the cucumber. In traditional Asian medicine, it’s used to promote longevity. Today’s scientists have discovered why Asian doctors prescribed G. pentaphyllum to address age-related health issues: It promotes AMPK activation.
G. pentaphyllum not only activates AMPK, but it also shuttles excess fats into the mitochondria to be utilized for energy and safe disposal. The result is efficient energy production and a sharp reduction in unnecessary fat storage.
Results of G. pentaphyllum-induced AMPK activation include increased fat burning, as well as an increase in cellular glucose uptake. Extracts of G. pentaphyllum have other beneficial properties as well, including the ability to prolong cellular life in the face of stresses induced by oxidation, fat accumulation, and diabetes.
When scientists began exploring the benefits of G. pentaphyllum for AMPK activation, they turned to animal studies. What they found was that leaf extracts of G. pentaphyllum activate AMPK, resulting in reduced body weight gain and fat accumulation. In a preclinical study, obese mice supplemented with G. pentaphyllum showed impressive declines in markers associated with obesity and its related diseases.
In another study, this time using diabetic rats, three weeks of G. pentaphyllum supplementation resulted in improved glucose tolerance by 35% and reduced new glucose production in the liver by 29%, with a reduction in liver glycogen, the storage form of sugar.
These results show the enormous beneficial impact of reducing circulating sugar and fats in response to AMPK activation by G. pentaphyllum.
Human studies have confirmed what many of the researchers had found in the lab: G. pentaphyllum boosts AMPK activity and provides important longevity benefits.
compelling human study, type II diabetics who were not using
diabetic medications drank a tea made with G. pentaphyllum. The
results compared to controls were:
None of these findings should be surprising since the prescription drug metformin, which is an AMPK activator, produces many of these same benefits."http://www.lifeextension.com/magazine/2014/ss/ampk/page-01
Well it can survive with glucose, fructose and amino acids.... So what's left?
Fat. What else is associated with that? Fasting—which leads to ketosis which leads to mTOR inhibition, AMPK activation, autophagy as well as p53, sirt1, foxo and a whole bunch of other anti cancer stuff.
I would basically dry fast and hold deep level ketosis (160) along with KETOGENIC diet until I killed it.
I would also employ longevity herbs or agents that inhibited mTOR, activated AMPK and promoted autophagy like what's in Interstellar Blend— Gynostemma, Reishi, Ginseng, Astragalus, Rhodiola, He Shou Wu etc the drugs Metformin and Rapamycin would be of interest as well.
I would definitely NOT be eating fruit or any glucose or fructose containing foods; I would also be VERY careful on protein; in fact I might just nix all 3 until cancer aborted. Fat only diet. Coconut oil a few times a day might do the trick and provide required energy aside from body fat reserves.
I would also use baking soda or milk of magnesia to keep my body ph alkaline while deep level ketosis.
nitric oxide levels high I would employ neo40 or arugula.
You conquer fear by facing it.
A dry fast is the conquering of fear. Fear of what? Fear of going without food, fear of going without water, without pills, without vitamins, supplements, weed, nicotine, caffeine, alcohol, drugs....whatever.
That's right, fear of the unknown. Fear of death. Fear of losing control. Fear of breaking. Fear of being vulnerable. Fear of failing. Fear of being revealed.
To the timid this seems like insanity.
No training wheels, filters, buffers, numbing agents, masks, props or diversions— just YOU and nothing else—nowhere to run and nothing to hide behind.
The body a sealed vessel....
THIS is how true strength of character and true strength of will is forged.
No mere mortals dry fast.
These ARE no ordinary human beings.
These are EXTRAORDINARY BEINGS.
"No water or food you say?"